Alice S. Huang, California Institute of Technology
Biology
Professor
Pasadena, California
alice.huang@caltech.edu
Office:
(626) 395-3446
Expert In
Bio/Research
Dr. Huang is Senior Faculty Associate in Biology at the California Institute of Technology. She was previously Professor of Microbiology and Molecular Genetics at Harvard Medical School, and subsequently Dean for Science at New York University. She became full Professor at Harvard in 1979, as we...
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Bio/Research
Dr. Huang is Senior Faculty Associate in Biology at the California Institute of Technology. She was previously Professor of Microbiology and Molecular Genetics at Harvard Medical School, and subsequently Dean for Science at New York University. She became full Professor at Harvard in 1979, as well as the Director of the Laboratories of Infectious Diseases at Children's Hospital in Boston.
The American Society for Microbiology awarded her the Eli Lilly Award in Immunology and Microbiology (1977) and the Alice C. Evans Award (2001). She is a fellow of the Academia Sinica in Taiwan, American Women in Science, the Academy of Microbiology, and the AAAS. Recently, she was elected the President of AAAS for 2010-11.
Her work, VSV, has become a model virus for many research studies. She was, also, the first to demonstrate that RNA and DNA enveloped viruses, including retroviruses, VSV, HIV and HSV, phenotypically mix their surface glycoproteins resulting in pseudotypes with alterations of antigenicity and host range.
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The American Society for Microbiology awarded her the Eli Lilly Award in Immunology and Microbiology (1977) and the Alice C. Evans Award (2001). She is a fellow of the Academia Sinica in Taiwan, American Women in Science, the Academy of Microbiology, and the AAAS. Recently, she was elected the President of AAAS for 2010-11.
Her work, VSV, has become a model virus for many research studies. She was, also, the first to demonstrate that RNA and DNA enveloped viruses, including retroviruses, VSV, HIV and HSV, phenotypically mix their surface glycoproteins resulting in pseudotypes with alterations of antigenicity and host range.
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